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Cold sores are the most common recurrent viral infection that people get-and University of Utah medical researchers say there's a good chance that a predisposition to them is inherited through one of six genes.
Children who possess a gene that allows them to produce high levels of nitric oxide are protected from two of the deadliest forms of malaria, according to a report published today.
An international consortium of researchers has identified the mutated gene that causes hyperparathyroidism-jaw tumor syndrome, a rare condition that leads to benign tumors of the parathyroid glands, jaw bones, and kidney. Affected people are also at risk of developing parathyroid cancer.
As people age, their telomeres-the ends of their chromosomes--get shorter, which holds major implications for people over 60 because shortened telomeres in blood are associated with increased risks of dying from heart disease or infectious diseases.
University of Utah School of Medicine infectious diseases specialists have made important findings regarding the production of nitric oxide and the ability of the human body to prevent or fight malaria, a major killer of children in tropical countries.
University of Utah researchers have developed a rapid direct sequence gene test that detects the most common form of muscular dystrophy in at least 95 percent of cases-a far higher success rate than the currently available test-and at a feasible cost.
A form of the hormone progesterone called hydroxyprogesterone caproate, or 17P, can reduce preterm births by up to one-third, according to a study due out in the June 12 New England Journal of Medicine.
Previous estimates of the amount of neutron dose received by Hiroshima atomic-bomb survivors are essentially accurate and the existing standards for estimating risk for radiation-induced cancer need not be changed.
University of Utah researchers have identified genes that ensure nerves develop in the correct part of the brain so mice can roll their eyeballs sideways, wiggle their whiskers, pull their ears back and blink their eyelids. The genes likely help control human facial expressions.
University of Utah biochemists have discovered that the ability to smell foods is regulated by ADAR enzymes' interaction with the RNA interference pathway. Research with C. elegans, a tiny worm suggests that behavioral defects associated with ADAR genes are caused by abberant RNAi, not codon changes.
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